Mr. Grier’s interest in investigating his own disease goes far beyond what the average patient would do.
You see Mr. Grier
created an organization that registers symptomatic patients for tissue
collection and brain autopsies at the time of their death.
His main interest is to find formerly treated Lyme patients that have gone on to be diagnosed with either MS or dementia.
“Since 1975 when Lyme
disease was first described in the medical literature, it has always
been an assumption that the organism that causes Lyme disease mostly an
arthritic disease and easily eradicated with the traditional and current
treatment protocols of antibiotics.”
Explains Mr. Grier.
“The truth is no serology test or spinal fluid test can accurately
detect the sequestered infection within the human brain.
The only way to know for sure is to do brain autopsies, and look directly at the brain tissue with special dyes and stains.
Without special
immune-antibody stains developed by the NIH, the bacteria would remain
completely invisible under the microscope.’’
“Sadly no one is doing
this kind of Lyme research; oddly the medical community seems to be
strangely resistant to this kind of medical research and I’d like to
know why? ” asks Mr. Grier.
“Pathology is far more
definitive than assumptions, and much of our current understanding of
Lyme is based on a very flimsy foundation of facts most of which turned
out to be completely wrong.”
Something that Mr Grier points out in his talk are the ten facts about Lyme that the experts got wrong.
As examples, Mr. Grier
cites that shortly after Lyme disease was first described in 1975 but
before we knew what actually caused Lyme in 1981; that the public was
told many things as absolute facts about Lyme disease that all turned
out to be untrue.
Grier continues, “We
were told by the experts of the time that Lyme was only transmitted by a
new species of tick found in the NE USA (Ixodes dammini discovered by
Andrew Spielman of Harvard) so Lyme was supposed to be a regional and
isolated illness.
We were told it was mainly an arthritic disease and it turns out it can cause severe neurological damage.
We were told it was not transmitted transplacentally but several fetal autopsies have dispelled that myth.
We were told by some Lyme experts that the Lyme disease rash has to be the size of a basketball or it isn’t Lyme disease.
The truth is many Lyme rashes are only a few centimeters or not even seen.
We were told that the
Lyme organism isn’t an intracellular organism which can help infections
hide and remain dormant and safe from the immune system.
But as it turns out
Lyme disease most definitely is an intracellular disease of the brain
and we have local brain autopsies that prove this to be true despite
their being treated aggressively with antibiotics.”
“The basis of our work
is the idea that the bacteria enters the brain early in the infection
and is trapped in brain tissue and even trapped inside individual brain
neurons.
This happens after the
protective barrier called the blood brain barrier is broken down by the
infection in the first two weeks before the Lyme tests can even detect
the infection.
Then after the
infection has been cleared from the blood stream either by our immune
system or by antibiotics, the blood brain barrier reseals itself weeks
later trapping the infection within the brain.”
Mr. Grier explains that the end result is that the immune system stops making antibodies that the Lyme tests are looking for.
All our Lyme tests are indirect tests and have many many pitfalls.
The infection in the brain remains relatively silent for years or even decades until it results in an MS-like condition.
Explains Mr. Grier, “It
isn’t a matter of if this happens because we already have individual
pathologies that reveal this to be true.
Following these
patients for years doing frequent brain MRIs reveals long-term treatment
results in a shrinking of the white-matter lesions that look similar to
MS lesions in the brain.
The question is how
often is it occurring and what kind of treatment strategies do we need
to create to detect brain involvement earlier, and how do we best treat
longstanding spirochetal infections within the human brain?”
One of the reasons
Mr. Grier has decided to do this talk in Hermantown was because of a
Lyme disease documentary being filmed in Twig MN by local Duluth
videographer Ben Barneveld who uncovered numerous disabled patients in
the local area with a history of Lyme disease.
Unexpectedly a large percentage of these treated Lyme patients progressed on after treatment to having such conditions as:
MS, ALS, Parkinson’s, Rheumatoid Arthritis, and enlarged hearts.
Most of these patients
only became aware that their neighbors were also sick like themselves,
after local Lyme patients organized a showing of a Lyme disease
documentary at the Twig town hall.
Now several of those
patients from the Twig-Hermantown area have since registered for brain
autopsies and have authorized their remains for Lyme disease research.
The lecture is titled: “Lyme On The Brain” and is a talk based almost entirely on autopsy data and pathology.
Mr. Grier’s lecture is
FREE and open to the general public
Saturday, April 28th
10 AM
Grace Lutheran Church
5600 Miller Trunk Highway
HERMANTOWN, MINN.
All questions should be directed to
Tom Grier 218-728-3914
2000 (Poland): Lyme borreliosis and Multiple sclerosis: Any Connection? PDF here
A Seroepidemic study. Ann Agric Environ Med. issue 7, 141-143
Synopsis:
10 out of
26 MS patients tested positive for Lyme borreliosis. Notes how it is
virtually impossible to make a distinction between late stage Lyme
disease and Multiple sclerosis, not even with MRI. Diagnosis of MS vs.
late stage neuroborreliosis are guesswork – there are no reliable tests
for either. Conclusion: Multiple sclerosis may often be associated with
Borrelia infection.
Ann Agric Environ Med. 2000;7(2):141-3.
Lyme borreliosis and multiple sclerosis: any connection? A seroepidemic study.
Source
Department of Occupational Biohazards, Institute of Agricultural Medicine, Jaczewskiego 2, 20-090 Lublin, Poland.
Abstract
A total of 769 adult
neurological patients hospitalised in clinics and hospitals situated in
the Lublin region (eastern Poland) were examined during the years
1997-2000 with ELISA test for the presence of anti-Borrelia burgdorferi
sensu lato antibodies. A statististically significant (p=0.0422)
relationship was found between the clinically confirmed diagnosis of
multiple sclerosis and the positive serologic reaction with Borrelia
antigen. Ten out 26 patients with multiple sclerosis (38.5%) showed
positive serologic reaction to Borrelia, whereas among the total number
of examined neurological patients the frequency of positive findings was
twice as low (19.4%). The result suggests that multiple sclerosis may
be often associated with Borrelia infection
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